Depression Treatment: The Effects of Qigong on Anxiety, Depression, and Psychological Well-Being: A Systematic Review and Meta-Analysis.

The effects of qigong on anxiety, depression, and psychological well-being: a systematic review and meta-analysis.

Filed under: Depression Treatment

Evid Based Complement Alternat Med. 2013; 2013: 152738
Wang F, Man JK, Lee EK, Wu T, Benson H, Fricchione GL, Wang W, Yeung A

Introduction. The effect of Qigong on psychological well-being is relatively unknown. This study systematically reviewed the effects of Qigong on anxiety, depression, and psychological well-being. Methods. Using fifteen studies published between 2001 and 2011, a systematic review was carried out and meta-analyses were performed on studies with appropriate homogeneity. The quality of the outcome measures was also assessed. Results. We categorized these studies into three groups based on the type of subjects involved as follows: (1) healthy subjects, (2) subjects with chronic illnesses, and (3) subjects with depression. Based on the heterogeneity assessment of available studies, meta-analyses were conducted in three studies of patients with type II diabetes in the second group, which suggested that Qigong was effective in reducing depression (ES = -0.29; 95% CI, -0.58-0.00) and anxiety (ES = -0.37; 95% CI, -0.66-0.08), as measured by Symptom Checklist 90, and in improving psychological well-being (ES = -0.58; 95% CI, -0.91-0.25) as measured by Diabetes Specific Quality of Life Scale. Overall, the quality of research methodology of existing studies was poor. Conclusions. Preliminary evidence suggests that Gigong may have positive effects on psychological well-being among patients with chronic illnesses. However the published studies generally had significant methodological limitations. More high-quality studies are needed.
HubMed – depression


The potential association of later initiation of oral/enteral nutrition on euthyroid sick syndrome in burn patients.

Filed under: Depression Treatment

Int J Endocrinol. 2013; 2013: 707360
Pérez-Guisado J, de Haro-Padilla JM, Rioja LF, Derosier LC, de la Torre JI

Objective. The aim of this study was to determine if early initiation of oral/enteral nutrition in burn patients minimizes the drop in fT3 levels, reduces the potential for euthyroid sick syndrome (ESS), and shortens the length of hospital stay (LHS). Subjects and Methods. We retrospectively evaluated the statistical association of serum fT3, fT4, and TSH at the first (2nd-5th day) and second sample collection (9th-12th day) after the burn injury in 152 burn patients. Three groups were established depending on time of initiation of the oral/enteral nutrition: <24?h before the injury (Group 1), 24-48?h after the injury (Group 2), and >48?h after the injury (Group 3). Results. They were expressed as mean ± standard deviation. We found that LHS and the fT3 levels were statistically different in the 3 groups. The LHS (in days) was, respectively, in each group, 16.77 ± 4.56, 21.98 ± 4.86, and 26.06 ± 5.47. Despite the quantifiable drop in fT3, ESS was present only at the first sample collection (2.61 ± 0.92 days) in Group 3, but there was no group with ESS at the second sample collection (9.89 ± 1.01 days). Our data suggest that early initiation of nutritional supplementation decreases the length of hospitalization and is associated with decreasing fT3 serum concentration depression. Conclusion. Early initiation of oral/enteral nutrition counteracts ESS and improves the LHS in burn patients.
HubMed – depression


GABAB receptor-mediated frequency-dependent and circadian changes in synaptic plasticity modulate retinal input to the Suprachiasmatic Nucleus.

Filed under: Depression Treatment

J Physiol. 2013 Feb 11;
Moldavan MG, Allen CN

Light is the most important environmental signal that entrains the circadian clock located in hypothalamic suprachiasmatic nucleus (SCN). The retinohypothalamic tract (RHT) was stimulated to simulate the light intensity-dependent discharges of intrinsically photosensitive retinal ganglion cells projecting axons to the hypothalamus. Excitatory postsynaptic currents (EPSCs) were evoked by paired-pulse stimulation or by application of stimulus trains, and recorded from SCN neurons in rat brain slices. Initial release probability (Pr) and synaptic plasticity changes depended on the strength of GABA(B) receptor (GABA(B)R)-mediated presynaptic inhibition and could be different at the same GABA(B)R agonist concentration. Facilitation caused by frequency-dependent relief of GABA(B)R-mediated inhibition was observed when the initial Pr was decreased to less than 15% of control during strong activation of presynaptic GABA(B) receptors by (±)baclofen (10 ?M), GABA (? 2 mM) or by GABA uptake inhibitor nipecotic acid (? 5 mM). In contrast, short-term synaptic depression appeared during baclofen (10 ?M) application when initial Pr was greater than 30% of control. Block of 4-AP-sensitive K+ currents increased the amplitude and time constant of decay of evoked EPSCs (eEPSCs), and decreased the GABA(B)R-mediated presynaptic inhibition. The GABA(B) receptor antagonist CGP55845 (3 ?M) increased the eEPSCs amplitude 30% throughout the light-dark cycle. During light and dark phases the RHT inputs to 55% and 33% of recorded neurons, respectively, were under GABA(B) inhibitory control indicating that the tonic inhibition induced by local changes of endogenous GABA concentration contributes to the circadian variation of RHT transmitter release. We conclude that GABA(B)R-mediated presynaptic inhibition decreased with increasing frequency and broadening of presynaptic action potentials, and depended on the sensitivity of RHT terminals to GABA(B)R agonists, and diurnal changes of the extracellular GABA concentration around RHT axon terminals in the SCN.
HubMed – depression



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